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Compared with normolipidemic obese subjects, type-2 diabetic patients had a higher apoB100 concentration in very low-density lipoproteins (VLDL), intermediate-density lipoproteins (IDL), and low-density lipoproteins

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(LDL) (P < 0.005). Glabrata appear intrinsically resistant to fluconazole. Treatment significantly decreased total apoB100 production rate (P < 0.05), but only for VLDL (P < 0.05). tamsulosin hcl 04mg cap

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We concluded that Atorvastatin ( Lipitor ) improved lipid abnormalities

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in type-2 diabetic patients not only by increasing the clearance of apoB100-containing lipoproteins

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but also by decreasing impotence supplements VLDL production. ApoB100 metabolism was studied using stable isotopes and multicompartmental modeling.

Cerevisiae and similarly

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identified by mass spectrometry. Kinetic analysis sho an increase in the total apoB100 production rate (P < 0.005) related to VLDL apoB100 overproduction

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(P < 0.005). Patients were also characterized by a lower fractional catabolic rate (FCR) in VLDL (not significant) or IDL (P < 0.005)

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mainly related to a decrease in VLDL and IDL delipidation rate (P <

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0.005). Mass spectrometric fingerprint analysis of SDS-PAGE separated plasma membrane

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fractions combined with heterologous hyper-expression provides a convenient method for protein identification and functional evaluation of induced proteins, even in an organism where the genome sequence database is incomplete.. The rapid induction of a

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multidrug efflux pump and/or overproduction of lanosterol 14alpha-demethylase are mechanisms that could make C. Catabolism of LDL was also lower in diabetic patients (P < 0.05). CgCdr1p was also functionally

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overexpressed in S. Mass spectrometry of trypsin-digested peptide fragments sho that the induced protein band comprised the ATP binding cassette-type drug efflux transporter CgCdr1p.

Kinetics of apolipoprotein B100 (apoB100)-containing lipoproteins

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were determined before and after Atorvastatin ( Lipitor ) treatment and compared with data obtained in five normolipidemic volunteers. Fluconazole ( Diflucan ) exposure rapidly induced expression of a 169-kDa protein band in plasma membrane fractions of C. Identification of two proteins induced by exposure of the pathogenic fungus Stesha glabrata

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to fluconazole.Candida glabrata is an increasingly important cause of opportunistic fungal infection of humans and appears to be intrinsically resistant to the triazole antifungal fluconazole. Treatment normalized FCR in IDL and LDL (P < 0.05). Mass spectrometric peptide fingerprinting identified this band as lanosterol 14alpha-demethylase, the enzyme in the ergosterol biosynthesis pathway targeted by fluconazole. A 61-kDa protein band in the plasma membrane fraction from C.

However, the mechanisms responsible for reduced susceptibility to azole drugs are not understood. Effect of Atorvastatin ( Lipitor ) on apolipoprotein B100 containing lipoprotein

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metabolism in type-2 diabetes.Seven hypertriglyceridemic patients with type-2 diabetes were treated with Atorvastatin ( Lipitor ) (40 mg/day) for 2 months. Glabrata was also induced by fluconazole ( Diflucan ) exposure. Atorvastatin ( Lipitor ) treatment significantly decreased plasma triglycerides (P < 0.05), total and LDL cholesterol (P < 0.05), apoB100 in LDL, IDL, and VLDL (P < 0.05).

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